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PSD has a reported incidence of 18% to 33%, though it is commonly underdiagnosed due to overlapping symptoms between stroke and depression. [3] A comprehensive meta-analysis found that over half of stroke patients experience at least one episode of depression. [4] Various risk factors increase the likelihood of developing PSD, including: [3] [4 ...
The incidence of post-stroke depression peaks at 3–6 months and usually resolves within 1–2 years after the stroke, although a minority of patients can go on to develop chronic depression. The diagnosis of post-stroke depression is complicated by other consequences of stroke such as fatigue and psychomotor retardation – which do not ...
In people with acute stroke and hemiparesis, the disorder is present in 10.4% of patients. [4] Rehabilitation may take longer in patients that display pusher behaviour. The Copenhagen Stroke Study found that patients that presented with ipsilateral pushing used 3.6 weeks more to reach the same functional outcome level on the Barthel Index, than did patients without ipsilateral pushing.
And when stroke patients are taken to an emergency room, the first treatment doctors often consider is a clot-dissolving medicine called tPA, which can make brain bleeding worse.
Other symptoms that may occur during PEM include cognitive impairment, flu-like symptoms, pain, weakness, and trouble sleeping. [6] [4] Though typically cast as a worsening of existing symptoms, patients may experience some symptoms exclusively during PEM. [6] Patients often describe PEM as a "crash", "relapse", or "setback". [6]
Constructional apraxia is common after right parietal stroke and it continues after visuospatial symptoms have subsided. [5] Patients with posterior and parietal lobe lesions tend to have the most severe symptoms. [9] In Alzheimer's disease research, the AT8 antibody has proven to be an early indicator of tau protein pathology.