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The gastric folds (or gastric rugae) are coiled sections of tissue that exist in the mucosal and submucosal layers of the stomach. [1] They provide elasticity by allowing the stomach to expand when a bolus enters it. These folds stretch outward through the action of mechanoreceptors, which respond to the increase in pressure. [2]
Ménétrier disease is a rare, acquired, premalignant disease of the stomach characterized by massive gastric folds, excessive mucus production with resultant protein loss, and little or no acid production (achlorhydria). The disorder is associated with excessive secretion of transforming growth factor alpha (TGF-α). [1]
Gastric antral vascular ectasia (GAVE) is an uncommon cause of chronic gastrointestinal bleeding or iron deficiency anemia. [ 1 ] [ 2 ] The condition is associated with dilated small blood vessels in the gastric antrum , which is a distal part of the stomach . [ 1 ]
Gastric contents should not be seen passing through the pylorus because if it does, pyloric stenosis should be excluded and other differential diagnoses such as pylorospasm should be considered. The positions of superior mesenteric artery and superior mesenteric vein should be noted because altered positions of these two vessels would be ...
In the stomach, the epithelium is simple columnar, and is organised into gastric pits and glands to deal with secretion. [1] In the small intestine, epithelium is simple columnar and specialised for absorption. It is organised into plicae circulares and villi, and the enterocytes have microvilli.
Rugae folds behind the anterior teeth in the hard palate of the mouth. In anatomy, rugae (sg.: ruga) are a series of ridges produced by folding of the wall of an organ. [1]In general, rugae are a biological feature found in many organisms, serving purposes such as increasing surface area, flexibility, or structural support.
Focal, irregular and asymmetrical gastrointestinal wall thickening on CT scan suggests a malignancy. [9] Segmental or diffuse gastrointestinal wall thickening is most often due to ischemic, inflammatory or infectious disease. [9] Though less common, medications such as ACE inhibitors can cause angioedema and small bowel thickening. [10]
Muscular EG (13–70%) present with obstruction of gastric outlet or small intestine; sometimes as an obstructing caecal mass or intussusception. Subserosal EG (4.5% to 9% in Japan and 13% in the US) [ 14 ] presents with ascites which is usually exudative in nature, abundant peripheral eosinophilia, and has favourable responses to corticosteroids .