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Hypercapnia also stimulates vasodilation of cerebral blood vessels, increased cerebral blood flow and elevated ICP presumably leading to headache, visual disturbance and other central nervous system (CNS) symptoms. CO 2 is a known potent vasodilator and an increase in cerebral perfusion pressure will increase the CSF production by about 4%. [17]
Intraocular pressure varies throughout the night and day. The diurnal variation for normal eyes is between 3 and 6 mmHg and the variation may increase in glaucomatous eyes. During the night, intraocular pressure may not decrease [17] despite the slower production of aqueous humour. [18]
The danger of g-LOC to aircraft pilots is magnified because on relaxation of g-force there is a period of disorientation before full sensation is re-gained. A g-suit does not so much increase the g-threshold, but makes it possible to sustain high g longer without excessive physical fatigue.
Under negative g, blood pressure will increase in the head, running the risk of the dangerous condition known as redout, with too much blood pressure in the head and eyes. Because of the high level of sensitivity that the eye’s retina has to hypoxia, symptoms are usually first experienced visually.
The efferent portion is carried by the vagus nerve from the cardiovascular center of the medulla to the heart, of which increased stimulation leads to decreased output of the sinoatrial node. [3] This reflex is especially sensitive in neonates and children, particularly during strabismus correction surgery . [ 4 ]
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Ocular hypertension is the presence of elevated fluid pressure inside the eye (intraocular pressure), usually with no optic nerve damage or visual field loss. [1] [2]For most individuals, the normal range of intraocular pressure is between 10 mmHg and 21 mmHg. [3]
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