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Cortical stimulation mapping led to the development of a homunculus for the motor and sensory cortices, which is a diagram showing the brain's connections to different areas of the body. An example is the cortical homunculus of the primary motor cortex and the somatosensory cortex, which are separated by the central sulcus.
Cerebellar ataxia is a form of ataxia originating in the cerebellum. [1] Non-progressive congenital ataxia (NPCA) is a classical presentation of cerebral ataxias.. Cerebellar ataxia can occur as a result of many diseases and may present with symptoms of an inability to coordinate balance, gait, extremity and eye movements. [2]
Ataxia (from Greek α- [a negative prefix] + -τάξις [order] = "lack of order") is a neurological sign consisting of lack of voluntary coordination of muscle movements that can include gait abnormality, speech changes, and abnormalities in eye movements, that indicates dysfunction of parts of the nervous system that coordinate movement, such as the cerebellum.
Somatotopagnosia has been argued to be a better suited term to describe the condition. While autotopagnosia emphasizes the deficiencies in localizing only one's own body parts and orientation, somatotopagnosia also considers the inability to orient and recognize the body parts of others or representations of the body (e.g., manikins, diagrams).
cortex, outer region Latin cortex, bark of a tree corticosteroid: cost-of or pertaining to the ribs: Latin costa, rib costochondral: cox-of or relating to the hip, haunch, or hip-joint Latin coxa, hip coxopodite: crani(o)-belonging or relating to the cranium: Latin cranium, from Greek κρᾱνίον (krāníon), cranium, skull, bones enclosing ...
Polymicrogyria (PMG) is a condition that affects the development of the human brain by multiple small gyri creating excessive folding of the brain leading to an abnormally thick cortex. This abnormality can affect either one region of the brain or multiple regions.
The spreading of a self-propagating wave of cellular depolarization in the cerebral cortex. The spreading of a wave of ischemia passing through an area of cortex. The spreading of a wave of vasoconstriction following vasodilation and prolonged sustained vasoconstriction of contiguous cortical arterioles. [12]
Seizures in FCD are likely caused by abnormal circuitry induced by the presence of DNs and BCs. These abnormal cell types generate abnormal electrical signals which spread out to affect other parts of the cerebral cortex. Medication is used to treat the seizures that may arise due to cortical dysplasia.