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Despite most dissociatives' main mechanism of action being tied to NMDA receptor antagonism, some of these substances, which are nonselective in action and affect the dopamine [2] and/or opioid [3] systems, may be capable of inducing more direct and repeatable euphoria or symptoms which are more akin to the effects of typical "hard drugs" or ...
Psychoactive substances that cause temporary dissociation tend to be NMDA receptor antagonists or Κ-opioid receptor agonists. [53] Although, this is not necessarily always the case and dissociation can occur with non-hallucinogenic drugs. [54]
Cause: The cause of dissociative identity disorder is contentious; it is most often considered to be caused either by ongoing childhood trauma that occurs before the ages of six to nine, [9] [10] or as an unintentional product of therapy, fantasy, or other sociogenic factors. [11] Treatment: Long-term psychotherapy to improve the patient's ...
The three main types of dissociative disorders are: Dissociative identity disorder. Once known as multiple personality disorder, dissociative identity disorder usually comes on after extreme abuse ...
Substance-induced psychosis (commonly known as toxic psychosis or drug-induced psychosis) is a form of psychosis that is attributed to substance intoxication, withdrawal or recent consumption of psychoactive drugs. It is a psychosis that results from the effects of various substances, such as medicinal and nonmedicinal substances, legal and ...
Ketamine is a sedative and it can cause dissociation—that is, a disconnect from your body, mind, and reality, like a drug trip. It has the potential for abuse, too. Ketamine is also known as the ...
The diagnosis of depersonalization disorder can be made with the use of the following interviews and scales: The Structured Clinical Interview for DSM-IV Dissociative Disorders (SCID-D) is widely used, especially in research settings. This interview takes about 30 minutes to 1.5 hours, depending on individual's experiences.
The neurological cause of psychogenic amnesia is controversial. [5] [7] Even in cases of organic amnesia, where there is lesion or structural damage to the brain, caution must still be taken in defining causation, as only damage to areas of the brain crucial to memory processing is possible to result in memory impairment. [7]