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Early dumping syndrome symptoms may include: [1] nausea; vomiting; abdominal pain and cramping; diarrhea; feeling uncomfortably full or bloated after a meal; sweating; weakness; dizziness; flushing, or blushing of the face or skin; rapid or irregular heartbeat; The symptoms of late dumping syndrome may include: [1] hypoglycemia; flushing
Symptoms of delayed gastric emptying tend to be exacerbated by eating, particularly after fatty foods and indigestible solids like salads and leafy vegetables. [10] In general, nausea is the most commonly reported symptom, affecting up to 96% of gastroparesis patients.
The gastric bypass group had an average peak alcohol breath level of 0.08%, whereas the control group had an average peak alcohol breath level of 0.05%. It took an average of 108 minutes for the gastric bypass patients group to return to an alcohol breath of zero, while it took the control group an average of 72 minutes. [journal 15]
Hyperinsulinism-hyperammonemia syndrome (HIHA) due to glutamate dehydrogenase 1 gene. Can cause intellectual disability and epilepsy in severe cases. [3] Gastric dumping syndrome (after gastrointestinal surgery) Other congenital metabolic diseases; some of the common include Maple syrup urine disease and other organic acidurias
Early dumping syndrome (emptying within 1 hour of eating) is also associated with a rapid drop in blood pressure, which may cause fainting. [48] Late dumping syndrome is characterized by low blood sugar 1–3 hours after a meal, presenting with palpitations, tremors, sweating, a feeling of faintness, and irritability. [48]
Bowel-associated dermatosis–arthritis syndrome (BADAS), is a complication of jejunoileal bypass surgery consisting of flu-like symptoms (fever, malaise), multiple painful joints (polyarthralgia), muscle aches and skin changes. It has been reported to occur in up to 20% of patients who had jejunoileal bypass surgery, a form of obesity surgery ...
Delay in the diagnosis of SMA syndrome can result in fatal catabolysis (advanced malnutrition), dehydration, electrolyte abnormalities, hypokalemia, acute gastric rupture or intestinal perforation (from prolonged mesenteric ischemia), gastric distention, spontaneous upper gastrointestinal bleeding, hypovolemic shock, and aspiration pneumonia.
Many patients are diagnosed late in the course of disease after additional symptoms are seen. Mortality is also difficult to accurately determine. One retrospective study estimated mortality to be between 10 and 25% for chronic intestinal pseudo-obstruction (CIPO) and to vary greatly depending on the etiology of the condition. [5]