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Generally, diseases outlined within the ICD-10 codes K50-K52 within Chapter XI: Diseases of the digestive system should be included in this category. Subcategories This category has the following 2 subcategories, out of 2 total.
Generally, diseases outlined within the ICD-10 codes A00-A09 within Chapter I: Certain infectious and parasitic diseases should be included in this category. Subcategories This category has the following 3 subcategories, out of 3 total.
Europe and other parts of the world use the ICD-10. The root codes for ICD-10 and ICD-10-CM are the same, making it helpful for locating codes for general body systems and disease processes. [2] [3] In ICD-11 the search and coding of any disease, including rare ones is done via the ICD-11 website. [4]
Enteritis is inflammation of the small intestine. It is most commonly caused by food or drink contaminated with pathogenic microbes, [ 1 ] such as Serratia , but may have other causes such as NSAIDs , radiation therapy as well as autoimmune conditions like coeliac disease .
Enterocolitis is an inflammation of the digestive tract, involving enteritis of the small intestine and colitis of the colon. [1] It may be caused by various infections, with bacteria, viruses, fungi, parasites, or other causes.
Eosinophilic gastroenteritis (EG or EGE), also known as eosinophilic enteritis, [1] is a rare and heterogeneous condition characterized by patchy or diffuse eosinophilic infiltration of gastrointestinal (GI) tissue, first described by Kaijser in 1937.
Vascular, bronchial and parenchymal margins of a lobectomy, showing staple line of bronchial margin being removed with scissors. Lobectomy means surgical excision of a lobe . This may refer to a lobe of the lung [ 1 ] (also simply called a lobectomy ), a lobe of the thyroid ( hemithyroidectomy ), a lobe of the brain (as in anterior temporal ...
Because individuals with LRBA deficiency usually have lower levels of CTLA-4 than those with CTLA-4 haploinsufficiency, the condition frequently presents earlier in life. [39] CTLA-4 haploinsufficiency results in abnormal regulatory T cell function and T cell proliferation, which leads to immunodeficiency and autoimmunity traits. [ 40 ]