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  2. Subepithelial connective tissue graft - Wikipedia

    en.wikipedia.org/wiki/Subepithelial_connective...

    The connective tissue is generally taken from the hard palate, although it may be taken from other sites as well, such as the maxillary tuberosity area. Because the connective tissue for the graft is transplanted without the superficial epithelium from the donor site, it is termed subepithelial.

  3. What to Know Before, During, and After Your Gum Graft Surgery

    www.aol.com/news/know-during-gum-graft-surgery...

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  4. Gingival grafting - Wikipedia

    en.wikipedia.org/wiki/Gingival_grafting

    Gum grafting, also known as a gingival graft or periodontal plastic surgery, is a surgical procedure to reverse gum recession. Gum recession exposes the roots of teeth, [9] which can lead to sensitivity and put teeth at a higher risk of damage or disease [10] due to the loosening of their attachment within the gums and bones of the jaw.

  5. Gingival recession - Wikipedia

    en.wikipedia.org/wiki/Gingival_recession

    Gingival recession, also known as gum recession and receding gums, is the exposure in the roots of the teeth caused by a loss of gum tissue and/or retraction of the gingival margin from the crown of the teeth. [1] Gum recession is a common problem in adults over the age of 40, but it may also occur starting in adolescence, or around the age of 10.

  6. Guided bone and tissue regeneration (dentistry) - Wikipedia

    en.wikipedia.org/wiki/Guided_bone_and_tissue...

    A study used e-PTFE membranes to cover surgically constructed average size bone defects in the mandibular angles of rats. Consequently, the e-PTFE membrane acted as a barrier to soft tissue and sped up bone healing, which took place between 3–6 weeks while no healing occurred in the non-membrane control group during a 22 week period. [16]

  7. Healing of periapical lesions - Wikipedia

    en.wikipedia.org/wiki/Healing_of_periapical_lesions

    In 1890, W.D. Miller, considered the father of oral microbiology, was the first to associate pulpal disease with the presence of bacteria. [11] This was confirmed by Kakehashi, who, in 1965, proved that bacteria were the cause of pulpal and periradicular disease in studies using animal models; pulpal exposures were initiated in both normal and germ-free rats, and while no pathologic changes ...

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