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Fibers from the optic nerves cross over in the optic chiasm with some fibers passing to the contralateral optic nerve tract. This is the basis of the "swinging-flashlight test". Loss of accommodation and continued pupillary dilation can indicate the presence of a lesion on the oculomotor nerve.
Oculomotor nerve palsy or oculomotor neuropathy [1] is an eye condition resulting from damage to the third cranial nerve or a branch thereof. As the name suggests, the oculomotor nerve supplies the majority of the muscles controlling eye movements (four of the six extraocular muscles, excluding only the lateral rectus and superior oblique).
V 3 (mandibular nerve) is located in the foramen ovale. Receives sensation from the face, mouth and nasal cavity, and innervates the muscles of mastication. VI Abducens: Mainly motor Nuclei lying under the floor of the fourth ventricle Pons. Located in the superior orbital fissure. Innervates the lateral rectus, which abducts the eye. VII Facial
The Edinger–Westphal nucleus also called the accessory or visceral oculomotor nerve, is one of the two nuclei of the oculomotor nerve (CN III) located in the midbrain. [1] [2] [3] It receives afferents from both pretectal nuclei (which have in turn received afferents from the optic tract). [4]
Unilateral lesions of the PPRF produce characteristic findings: Loss of horizontal saccades directed towards the side of the lesion, no matter the current position of gaze; Contralateral gaze deviation (acute lesions, such as early stroke, only) Gaze-evoked lateral nystagmus on looking away from the side of the lesion
Specifically, compression or ischemic damage of the mesencephalic tectum, including the superior colliculus adjacent oculomotor (origin of cranial nerve III) and Edinger-Westphal nuclei, causing dysfunction to the motor function of the eye. Classically, it has been associated with three major groups:
An alternative anatomical cause is a lesion of the abducens nucleus (VI) on one side (resulting in a failure of abduction of the ipsilateral eye and adduction of the contralateral eye = conjugate gaze palsy towards affected side), with interruption of the ipsilateral medial longitudinal fasciculus after it has crossed the midline from its site ...
The cranial nerves and cranial nerve nuclei are also located in the brainstem making them susceptible to damage from a brainstem lesion. Cranial nerves III (Oculomotor), VI (Abducens), and XII (Hypoglossal) are most often associated with this syndrome given their close proximity with the pyramidal tract, the location which upper motor neurons ...