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  2. 5 years since COVID declared public health emergency in US ...

    www.aol.com/5-years-since-covid-declared...

    "The virus is still persistent and still moving. It's still not a trivial thing," Clarke told ABC News. "It hasn't vanished, it's just at a different point." PHOTO: In this Jan. 31, 2020, file ...

  3. Public health experts are warning of a ‘quad-demic’ this ...

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    Public health experts are warning of a ‘quad-demic’ this winter. Here’s where flu, COVID, RSV, and norovirus are spreading

  4. Is COVID finally gone? What to know about the virus 4 years ...

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    Here’s what you should know about COVID in 2024. The most common Covid symptoms are consistent with the most recent dominant variant, JN.1. Coughing

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  6. RSV may be leveling off, but flu cases are still ... - AOL

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    After an unusually early spike in respiratory viruses, RSV may have peaked or leveled off in some parts of the U.S., while influenza and COVID-19 cases are climbing.

  7. COVID-19 pandemic - Wikipedia

    en.wikipedia.org/wiki/COVID-19_pandemic

    In early and mid-January, the virus spread to other Chinese provinces, helped by the Chinese New Year migration. Wuhan was a transport hub and major rail interchange. [204] On 10 January, the virus' genome was shared publicly. [205] A retrospective study published in March found that 6,174 people had reported symptoms by 20 January. [206]

  8. Virus latency - Wikipedia

    en.wikipedia.org/wiki/Virus_latency

    Virus latency (or viral latency) is the ability of a pathogenic virus to lie dormant within a cell, denoted as the lysogenic part of the viral life cycle. [1] A latent viral infection is a type of persistent viral infection which is distinguished from a chronic viral infection. Latency is the phase in certain viruses' life cycles in which ...

  9. SARS-CoV-2 - Wikipedia

    en.wikipedia.org/wiki/SARS-CoV-2

    Virus infections start when viral particles bind to host surface cellular receptors. [158] Protein modeling experiments on the spike protein of the virus soon suggested that SARS‑CoV‑2 has sufficient affinity to the receptor angiotensin converting enzyme 2 (ACE2) on human cells to use them as a mechanism of cell entry. [159]