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Kawasaki disease (also known as mucocutaneous lymph node syndrome) is a syndrome of unknown cause that results in a fever and mainly affects children under 5 years of age. [6] It is a form of vasculitis , in which medium-sized blood vessels become inflamed throughout the body. [ 1 ]
Tomisaku Kawasaki (川崎 富作, Kawasaki Tomisaku, February 1, 1925 – June 5, 2020) was a Japanese pediatrician who first described the condition now known as Kawasaki disease in the 1960s. [ 1 ] [ 2 ] Alongside rheumatic heart disease , Kawasaki disease is considered to be the leading cause of acquired heart disease in children worldwide.
Kawasaki disease is a type of vasculitis where the endothelial cells in the coronary arteries become damaged, potentially leading to complications like myocardial infarction and aneurysms. This video covers the known pathophysiology, important signs and symptoms, diagnostic criteria, and treatment.
Kawasaki disease is most commonly seen in infants and children under five years old and is more likely to affect boys. The disease is self-limited which means that the inflammation will resolve after 6 to 8 weeks but if we left it untreated, there is a 20-25% risk of the heart complications we went over. Alright so let’s look at the symptoms ...
Kawasaki disease (Kawasaki's), a vascular disease found primarily in young children; Kawasaki Racecourse, a horseracing dirt track, in Kawasaki, Kanagawa, Japan; Shaking rat Kawasaki, the Kawasaki lineage of laboratory rat animals; Kawasaki-type oiler (Japanese: 川崎型油槽船, romanized: Kawasaki-gata Yusōsen), an oil tanker and refueller ...
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The median age of onset appears to be at least 7 years (compared with 2 years for Kawasaki disease, which primarily affects children under the age of 5). [22] Male children seem to be more frequently affected (broadly in line with Kawasaki disease, where the male to female ratio is about 1.5 to 1). [22]
Coronary artery disease (atherosclerosis) Vasculitic and connective tissue diseases (Kawasaki and Marfan) Intracoronary manipulation leading to local wall stress (stent placement, angioplasty, brachytherapy) Post-infectious as a consequence of direct wall infiltration or immune complex deposition [8]