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With Atrial fibrillation, or A-fib or AF, signals move around the atria in a completely disorganized way that tends to override the sinus node. Instead of a one big contraction then, you get all these mini contractions that make it just look like the atria are just quivering.
This causes the refractory period of atrial tissue to increase, hence its effectiveness in the treatment of atrial fibrillation and atrial flutter. Dofetilide does not affect dV/dT max (the slope of the upstroke of phase 0 depolarization), conduction velocity, or the resting membrane potential.
Atrial fibrillation is associated with an increased risk of heart failure, dementia, and stroke. [3] [12] It is a type of supraventricular tachycardia. [14] Atrial fibrillation frequently results from bursts of tachycardia that originate in muscle bundles extending from the atrium to the pulmonary veins. [15]
This is the property of the AV node that prevents rapid conduction to the ventricle in cases of rapid atrial rhythms, such as atrial fibrillation or atrial flutter. The AV node's normal intrinsic firing rate without stimulation (such as that from the SA node) is 40–60 times/minute. [13]
For TIC due to atrial fibrillation, rate control, rhythm control, and RF catheter ablation can be effective to control the tachyarrhythmia and improve left ventricular systolic function. [ 5 ] [ 9 ] For TIC due to atrial flutter, rate control is often difficult to achieve, and RF catheter ablation has a relatively high success rate with a low ...
The electrical signal then travels through both the right and left atrium and causes the two atria to contract at the same time. This simultaneous contraction results in the P wave seen in an ECG tracing. [citation needed] The electrical signal then travels to the AV node located on the lower portion of the interatrial septum. At the AV node ...
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