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The ganglionated plexi (autonomic ganglia of the heart atrium and ventricles) can also be a source of atrial fibrillation, and is sometimes also ablated for that reason. [16] Not only the pulmonary vein, but the left atrial appendage and Ligament of Marshall can be a source of atrial fibrillation and are also ablated for that reason.
The types of SVT associated with TIC include atrial fibrillation, atrial flutter, incessant atrial tachycardia, permanent junctional reciprocating tachycardia, atrioventricular reciprocating tachycardia, and atrioventricular nodal reentry tachycardia. [1] Atrial fibrillation is the most common and well-studied etiology of TIC. [1] [5]
Traumatic cardiac arrest can occur in patients following any severe blunt or penetrating injury to the chest. Following the traumatic event, the heart ceases to pump blood through the body. Unlike medical cardiac arrest, there are several potentially reversible causes that may result in cardiac arrest in the setting of trauma.
Depending on the patient's health and other variables such as medications taken for rate control, atrial fibrillation may cause heart rates that span from 50 to 250 beats per minute (or even higher if an accessory pathway is present). However, new-onset atrial fibrillation tends to present with rates between 100 and 150 beats per minute. [22]
Fibrillation can affect the atrium (atrial fibrillation) or the ventricle (ventricular fibrillation): ventricular fibrillation is imminently life-threatening. Atrial fibrillation affects the upper chambers of the heart, known as the atria. Atrial fibrillation may be due to serious underlying medical conditions and should be evaluated by a ...
The most common cause of myocardial rupture is a recent myocardial infarction, with the rupture typically occurring three to five days after infarction. [3] Other causes of rupture include cardiac trauma, endocarditis (infection of the heart), [4] [5] cardiac tumors, infiltrative diseases of the heart, [4] and aortic dissection. [citation needed]
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