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  2. N-Acetyl-γ-aminobutyric acid - Wikipedia

    en.wikipedia.org/wiki/N-Acetyl-γ-aminobutyric_acid

    [2] [3] [4] The pathway is a minor pathway in GABA synthesis compared to the main pathway in which GABA is synthesized from glutamate. [ 2 ] [ 3 ] [ 4 ] However, the pathway has been found to have an important physiological role in the brain, for instance in the production of GABA in the striatum and resultant inhibition of dopaminergic neurons ...

  3. 4-Acetamidobutanal - Wikipedia

    en.wikipedia.org/wiki/4-Acetamidobutanal

    4-Acetamidobutanal, also known as N-acetyl-γ-aminobutyraldehyde, N-acetyl-GABAL, or N-acetyl-GABA aldehyde, is a metabolic intermediate in the biosynthesis of γ-aminobutyric acid (GABA) from putrescine.

  4. Everything You Need to Know About GABA, an Essential ... - AOL

    www.aol.com/lifestyle/everything-know-gaba...

    Here, experts explain GABA uses and side effects. Otherwise known as gamma-aminobutyric acid, GABA is an amino acid produced in the brain. Here, experts explain GABA uses and side effects.

  5. GABA receptor agonist - Wikipedia

    en.wikipedia.org/wiki/GABA_receptor_agonist

    Gamma-aminobutyric acid, a GABA-B receptor agonist. A GABA receptor agonist is a drug that is an agonist for one or more of the GABA receptors, producing typically sedative effects, and may also cause other effects such as anxiolytic, anticonvulsant, and muscle relaxant effects. [1] There are three receptors of the gamma-aminobutyric acid. The ...

  6. N-Acetyl-γ-aminobutyraldehyde - Wikipedia

    en.wikipedia.org/wiki/N-Acetyl-GABAL

    N-Acetyl-γ-aminobutyraldehyde (N-acetyl-GABAL), also known as N-acetyl-GABA aldehyde, is a metabolic intermediate in the biosynthesis of γ-aminobutyric acid (GABA) from putrescine.

  7. Semax - Wikipedia

    en.wikipedia.org/wiki/Semax

    Semax is a medication which is used in Russia and Eastern Europe for the treatment of a broad range of conditions like brain trauma but predominantly for its claimed nootropic, neuroprotective, and neurorestorative effects.

  8. γ-Acetylenic GABA - Wikipedia

    en.wikipedia.org/wiki/Γ-Acetylenic_GABA

    Like other GABA-T inhibitors, γ-acetylenic GABA causes GABA levels in the brain to be elevated. This is due to 4-aminobutyrate transaminase being the enzyme that converts γ-aminobutyric acid to L-glutamate. Inhibiting the enzyme stops this conversion from happening.

  9. Selank - Wikipedia

    en.wikipedia.org/wiki/Selank

    Selank has an advantage over traditional anxiety treatments, such as benzodiazepines and barbiturates, as it has no sedating or negative cognitive side effects and no associated addiction or withdrawal problems. [citation needed] Selank is closely related to another nootropic drug, Semax, also developed by the Institute of Molecular Genetics in ...