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Constructional apraxia is common after right parietal stroke and it continues after visuospatial symptoms have subsided. [5] Patients with posterior and parietal lobe lesions tend to have the most severe symptoms. [9] In Alzheimer's disease research, the AT8 antibody has proven to be an early indicator of tau protein pathology.
Gerstmann syndrome is a neurological disorder that is characterized by a constellation of symptoms [1] that suggests the presence of a lesion usually near the junction of the temporal and parietal lobes at or near the angular gyrus. Gerstmann syndrome is typically associated with damage to the inferior parietal lobule of the dominant hemisphere ...
Parietal lobe (red). Allochiria has been observed mainly in the context of neglect which is usually due to a lesion that affects the right parietal lobe. [4] In patients with allochiria, their sensibility is retained completely but the patient is not clear as to which side of the body has been touched.
For example, a stroke affecting the right parietal lobe of the brain can lead to neglect for the left side of the visual field, causing a patient with neglect to behave as if the left side of sensory space is nonexistent (although they can still turn left). In an extreme case, a patient with neglect might fail to eat the food on the left half ...
Visual extinction arises from damage to the parietal lobe of the brain. This damage most frequently arises following a stroke or other infarction in that area – however, any traumatic event sufficient to cause widespread tissue damage in the area may cause sufficient harm.
Amorphosynthesis is most closely related to damage of the right parietal lobe but instances of left parietal and bilateral damage have also been reported. [16] The inattention to or suppression of somatic sensations on the contralesional side of the body can manifest in the cerebral processing centers that produce the sensory modalities for touch, taste, vision, smell, and proprioception. [17]