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Urinary specific gravity: 1.003 [1] [2] 1.030 [1] [2] g/mL Urobilinogen: 0.2 [2] 1.0 [2] Ehrlich units or mg/dL Free catecholamines, dopamine: 90 [3] 420 [3] μg/d Red blood cells (RBCs) 0 [4] [2] 2 [2] - 3 [4] per High Power Field (HPF) RBC casts: n/a 0 / negative [2] White blood cells (WBCs) 0 [2] 2 [2] pH: 5 [2] 7 [2] (unitless) Protein: 0 ...
It normally ranges from 1.003 to 1.035; lower values indicate that the urine is dilute, while higher values mean that it is concentrated. A urine specific gravity that consistently remains around 1.010 (isosthenuria) can indicate kidney damage, as it suggests that the kidneys have lost the ability to control urine concentration. [39]
Increasing the urine pH to a value higher than 7.0 may increase the risk of calcium phosphate stone formation, though this concept is controversial since citrate does inhibit calcium phosphate crystallization. Testing the urine periodically with nitrazine paper can help to ensure the urine pH remains in this optimal range. Using this approach ...
[3] [4] Typically hyperkalemia does not cause symptoms. [1] Occasionally when severe it can cause palpitations, muscle pain, muscle weakness, or numbness. [1] [2] Hyperkalemia can cause an abnormal heart rhythm which can result in cardiac arrest and death. [1] [3] Common causes of hyperkalemia include kidney failure, hypoaldosteronism, and ...
For urine collected via bladder catheterization in men and women, a single urine specimen with greater than 100,000 colony forming units of a single species per millilitre is considered diagnostic. [16] The threshold for women displaying UTI symptoms can be as low as 100 colony forming units of a single species per millilitre.
The opposite state is called hyperkalemia that means high level of potassium in the blood serum. [1] The speed at which potassium should be replaced depends on whether or not there are symptoms or abnormalities on an electrocardiogram. [1] Potassium levels that are only slightly below the normal range can be managed with changes in the diet. [3]
It was this multistage test — a description of which was first published in 1963 — that became known as the Bruce Protocol. In the initial paper, Bruce reported that the test could detect signs of such conditions as angina pectoris, a previous heart attack, or a ventricular aneurysm. Bruce and his colleagues also demonstrated that exercise ...
Abnormal heart rhythms and asystole are possible complications of hypermagnesemia related to the heart. [8] Magnesium acts as a physiologic calcium blocker, which results in abnormalities of the electrical conduction system of the heart. [citation needed] Consequences related to serum concentration: [9]: 281 4.0 mEq/L – Decreased reflexes