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  2. Adalimumab - Wikipedia

    en.wikipedia.org/wiki/Adalimumab

    The best-selling drugs list published by Genetic Engineering & Biotechnology News, shows that Humira occupied the #1 position for 2015 (US$14.012 billion) and 2016 (US$16.078 billion) [99] From 2012 until the US patent expired in 2016, Humira led the list of top-selling pharmaceutical products, and in 2016, it had US$16 billion of global sales ...

  3. AbbVie may see less pain from Humira sales decline as ... - AOL

    www.aol.com/news/abbvie-may-see-less-pain...

    AbbVie Inc is likely to beat Wall Street sales estimates for Humira in the second quarter as the only copycat in the U.S. market for the blockbuster arthritis drug struggled to gain traction.

  4. Don't Blame AbbVie for Humira's Success - AOL

    www.aol.com/2013/03/11/dont-blame-abbvie-for...

    Flip to the 2012 financial results for AbbVie and you'll see that its best-selling drug, Humira, brought in $9.27 billion in 2012, or 51% of the company's total revenue. AndroGel, soon to be the ...

  5. Drugmaker AbbVie expects Humira volume erosion to worsen

    www.aol.com/news/abbvie-raises-annual-profit...

    AbbVie generated $2.27 billion in Humira sales in the first quarter, roughly in line with estimates of $2.28 billion.The drugmaker and its investors have focused on sales of Skyrizi and Rinvoq to ...

  6. Immunotherapy - Wikipedia

    en.wikipedia.org/wiki/Immunotherapy

    Immunotherapy or biological therapy is the treatment of disease by activating or suppressing the immune system.Immunotherapy is designed to elicit or amplify an immune response are classified as activation immunotherapies, while immunotherapies that reduce or suppress are classified as suppression immunotherapies.

  7. Immunosuppressive drug - Wikipedia

    en.wikipedia.org/wiki/Immunosuppressive_drug

    A TNF-α (tumor necrosis factor-alpha) binding protein is a monoclonal antibody or a circulating receptor such as infliximab (Remicade), etanercept (Enbrel), or adalimumab (Humira) that binds to TNF-α, preventing it from inducing the synthesis of IL-1 and IL-6 and the adhesion of lymphocyte-activating molecules.

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