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An open-label pilot study found that topical pimecrolimus cream helped dark-skinned patients with postinflammatory hypopigmentation related to seborrheic dermatitis. [ 8 ] When functional melanocytes are present in the damaged area, sun or ultraviolet (UV) exposure may aid in repigmentation; however, excessive exposure may intensify the color ...
Unusual conditions that can be associated with a lichenoid inflammatory cell infiltrate are HIV dermatitis, syphilis, mycosis fungoides, urticaria pigmentosa, and post-inflammatory hyperpigmentation. [2] In cases of post-inflammatory hyperpigmentation, it is important to exclude potentially harmful mimics such as a regressed melanocytic lesion ...
692.72 Solar radiation dermatitis; 692.75 Disseminated actinic porokeratosis; 692.8 Contact dermatitis and other eczema due to other specified agents. 692.81 Dermatitis, due to cosmetics; 692.83 Dermatitis, due to metals; 692.9 Contact dermatitis NOS; 693 Dermatitis due to substances taken internally. 693.0 Dermatitis due to drugs and medicines ...
Treatments for Hypopigmentation [8] Initial Cause of Discoloration: Treatment: Idiopathic guttate hypomelanosis: No treatment Postinflammatory hypopigmentation: Treat the underlying inflammatory disease to restore pigmentation Pityriasis versicolor: A topical ointment, such as selenium sulfide 2.5% or imidazoles.
Tinea versicolor (also pityriasis versicolor) is a condition characterized by a skin eruption on the trunk and proximal extremities. [1] The majority of tinea versicolor is caused by the fungus Malassezia globosa, although Malassezia furfur is responsible for a small number of cases.
Hyperpigmentation results from an increase in melanin synthesis, which is mostly brought on by sun exposure, dermatological disorders, hormones, aging, genetic factors, skin injuries or inflammation, and acne. Sun exposure, which greatly increases the synthesis of melanin, is the most common cause of hyperpigmentation.
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