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For example, in the context of depression, the diathesis-stress model can help explain why Person A may become depressed while Person B does not, even when exposed to the same stressors. [7] More recently, the diathesis-stress model has been used to explain why some individuals are more at risk for developing a disorder than others. [9]
The concept of Environmental Sensitivity integrates multiple theories on how people respond to negative and positive experiences. These include the frameworks of Diathesis-stress model [4] and Vantage Sensitivity, [5] as well as the three leading theories on more general sensitivity: Differential Susceptibility, [6] [7] Biological Sensitivity to Context, [8] and Sensory processing sensitivity ...
A related view, the diathesis-stress model, posits that mental disorders result from genetic dispositions and environmental stressors, combining to cause patterns of distress or dysfunction. [19] The model is one way to explain why some individuals are more vulnerable to mental disorders than others.
The diathesis-stress model is an interactionism approach. In the context of schizophrenia, diathesis is the vulnerability. Vulnerabilities can be either genetic predisposition or early experiences, or both. [22] Stressors are any event that can trigger a schizophrenia-vulnerable individual to the onset of the condition.
The idea that individuals vary in their sensitivity to their environment was historically framed in diathesis-stress [4] or dual-risk terms. [5] These theories suggested that some "vulnerable" individuals, due to their biological, temperamental and/or physiological characteristics (i.e., "diathesis" or "risk 1"), are more vulnerable to the adverse effects of negative experiences (i.e., "stress ...
The diathesis–stress model [23] emphasizes the importance of applying multiple causality to psychopathology, by stressing that disorders are caused by both precipitating causes, and predisposing causes. A precipitating cause is an immediate trigger that instigates a person's action or behavior.
[5] [37] The diathesis–stress model specifies that depression results when a preexisting vulnerability, or diathesis, is activated by stressful life events. The preexisting vulnerability can be either genetic , [ 38 ] [ 39 ] implying an interaction between nature and nurture , or schematic , resulting from views of the world learned in ...
The investigators suggested that in utero insults may specifically stress those with a schizophrenia diathesis, supporting a "diathesis-stress" etiological model for a subset of schizophrenia (that Kraepelin identified) with early abnormalities suggesting brain atrophy. [57]