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And the zombie cell-killing drugs didn’t seem to slow bone loss; it just improved bone creation rates. ... This research was the clinical trial to test a zombie cell-killer treatment in humans ...
The body removes most of them. But others linger like zombies. They aren't dead. But as the Mayo Clinic's Nathan LeBrasseur puts it, they can harm nearby cells like moldy fruit corrupting a fruit ...
Possible senolytic agents are under preliminary research, including some which are in early-stage human trials. [6] [7] [clarification needed] The majority of candidate senolytic compounds are repurposed anti-cancer molecules, such as the chemotherapeutic drug dasatinib and the experimental small molecule navitoclax.
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Natural killer cells directly kill senescent cells, and produce cytokines which activate macrophages which remove senescent cells. [65] Senescent cells can be phagocytized by neutrophils as well as by macrophages. [66] Senolytic drugs which induce apoptosis in senescent cells rely on phagocytic immune system cells to remove the apoptosed cells ...
Embryonic stem cells and germ cells have also been described as immortal. [8] [9] Immortal cell lines of cancer cells can be created by induction of oncogenes or loss of tumor suppressor genes. One way to induce immortality is through viral-mediated induction of the large T-antigen, [10] commonly introduced through simian virus 40 (SV-40). [11]
Overview of signal transduction pathways involved in apoptosis. Cell death is the event of a biological cell ceasing to carry out its functions. This may be the result of the natural process of old cells dying and being replaced by new ones, as in programmed cell death, or may result from factors such as diseases, localized injury, or the death of the organism of which the cells are part.
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