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Autophagy degrades damaged organelles, cell membranes and proteins, and insufficient autophagy is thought to be one of the main reasons for the accumulation of damaged cells and aging. [87] Autophagy and autophagy regulators are involved in response to lysosomal damage, often directed by galectins such as galectin-3 and galectin-8 .
Autophagy is a monthly peer-reviewed scientific journal covering all aspects of cell autophagy. It is published by Taylor & Francis and the editor-in-chief is Daniel J. Klionsky ( University of Michigan ).
A comprehensive review with over 1,500 citations by Deretic and colleagues summarizes the role of autophagy in immunity and inflammation: [4] A more recent review [5] by Deretic in the Cell Press journal Immunity summarizes the role of autophagy in inflammation and how it affects various diseases, from autoimmunity to cancer, infections ...
Cuervo, AM (13 July 2011). "Chaperone-mediated autophagy: Dice's 'wild' idea about lysosomal selectivity". Nature Reviews Molecular Cell Biology. 12 (8): 535– 41. doi:10.1038/nrm3150. PMID 21750569. S2CID 23128629. Kaushik, S; Cuervo, AM (2009). "Chapter 19 Methods to Monitor Chaperone-Mediated Autophagy". Autophagy in Mammalian Systems, Part ...
Programmed cell death (PCD; sometimes referred to as cellular suicide [1]) is the death of a cell as a result of events inside of a cell, such as apoptosis or autophagy. [2] [3] PCD is carried out in a biological process, which usually confers advantage during an organism's lifecycle.
Autophagy-related protein 8 (Atg8) is a ubiquitin-like protein required for the formation of autophagosomal membranes. The transient conjugation of Atg8 to the autophagosomal membrane through a ubiquitin -like conjugation system is essential for autophagy in eukaryotes .
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Atg1 is a kinase upregulated upon induction of autophagy. Atg13 regulates Atg1 and together they form a complex called Atg13:Atg1, which receives signals from the master of nutrient sensing – Tor. Atg1 is also important in late stages of autophagosome formation. [8]