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Frontal lobe disorder, also frontal lobe syndrome, is an impairment of the frontal lobe of the brain due to disease or frontal lobe injury. [5] The frontal lobe plays a key role in executive functions such as motivation, planning, social behaviour, and speech production.
Hypofrontality is a symptom of numerous neurological diseases defined as reduced utilization of glucose and blood flow in the prefrontal cortex. Hypofrontality can be difficult to detect under resting conditions, but under cognitive challenges, it has been seen to correlate with memory deficits along with executive function deficits.
Cognitive symptoms from steroids appear within the first few weeks of treatment, appear to be dose dependent, and may or may not be accompanied by steroid psychosis or other Cushing's-type symptoms. [4] The symptoms include deficits in verbal and non-verbal memory; working memory; attention; sustained concentration; executive function ...
The symptoms observed in bvFTD are caused by dysfunction of the orbitofrontal cortex; thus these two neuropsychological tests might be useful in detecting early-stage bvFTD. However, as self-monitoring and somatic marker processes are so complex, it likely involves other brain regions.
Attention deficit hyperactivity disorder (ADHD) [1] is a neurodevelopmental disorder characterized by executive dysfunction occasioning symptoms of inattention, hyperactivity, impulsivity and emotional dysregulation that are excessive and pervasive, impairing in multiple contexts, and developmentally-inappropriate.
In the dissociative subtype of PTSD, there is both excessive control of emotions through suppressed limbic structures and insufficient control of emotions in the hyperactivity of the medial prefrontal cortex. Increased activity in the medial prefrontal cortex is associated with non-dissociative symptoms such as re-experiencing and hyperarousal ...
Animated image showing prefrontal cortex, which is thought to play a role in DPDR. There is converging evidence that the prefrontal cortex may inhibit neural circuits that normally form the basis of emotional experience. [36] In an fMRI study of DPDR patients, emotionally aversive scenes activated the right ventral prefrontal cortex.
The anterior superior frontal lobe is known as the prefrontal cortex which is responsible for the initiation and ideation of verbal speech. [7] The damage leaves the major language networks, Broca's and Wernicke’s areas and the arcuate fasiculus , unaffected. [ 1 ]