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Other antimalarials like amodiaquine, halofantrine, and hydroxychloroquine have also been linked to pruritus, albeit less frequently and to a lesser extent. [2] Another class of medications known to occasionally cause itching is known as serotonin reuptake inhibitors. [3] Itching is one of the most frequent adverse effects of opioid therapy. [4]
Pruritus ani is the irritation of the skin at the exit of the rectum, known as the anus, causing the desire to scratch. [1] The intensity of anal itching increases from moisture, [2] pressure, and rubbing caused by clothing and sitting. At worst, anal itching causes intolerable discomfort that often is accompanied by burning and soreness.
TMP/SMX is commonly used due to its ability to achieve high concentrations in urinary tract tissues and urine. This antibiotic combination demonstrates notable efficacy in both the treatment and prophylaxis of recurrent urinary tract infections. [12] Common adverse effects include nausea, vomiting, rash,pruritus, and photosensitivity. [26]
[14] [11] [20] On the other hand, the findings of animal studies of methenamine and carcinogenicity have also been questioned by some authors, [20] though this topic is controversial. [11] Antibiotics are known to disrupt the gut, urinary tract, and vaginal microbiota. [14] This has been associated with increased risk of recurrent UTIs. [14]
Aerococcus urinae is a member of the bacterial genus Aerococcus.The bacterium is a Gram-positive, catalase-negative coccus growing in clusters. Isolates of this genus were originally isolated in 1953 from samples collected in the air and dust of occupied rooms and were distinguished by their tetrad cellular arrangements. [2]
Xerosis is a contributing factor to pruritus and is present in 50–85% of patients with uremic pruritus. [16] Many studies have shown that emollients such as glycerol and paraffin, [17] physiological lipids, [18] 10% urea and dexpanthenol, [19] and baby oil [20] can reduce xerosis and pruritus in patients with uremic pruritus.
Swimmer's itch was known to exist as early as the 19th century, but it was not until 1928 that a biologist found that the dermatitis was caused by the larval stage of a group of flatworm parasites in the family Schistosomatidae. [7] The genera most commonly associated with swimmer's itch in humans are Trichobilharzia [4] and Gigantobilharzia.
692.3 Contact dermatitis and other eczema due to drugs and medicines in contact with skin; 692.4 Contact dermatitis and other eczema due to other chemical products; 692.5 Contact dermatitis and other eczema due to food in contact with skin; 692.6 Contact dermatitis, due to plants; 692.7 Contact dermatitis and other eczema due to solar radiation ...