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The differential susceptibility theory proposed by Jay Belsky [1] is another interpretation of psychological findings that are usually discussed according to the diathesis-stress model. Both models suggest that people's development and emotional affect are differentially affected by experiences or qualities of the environment.
The concept of Environmental Sensitivity integrates multiple theories on how people respond to negative and positive experiences. These include the frameworks of Diathesis-stress model [4] and Vantage Sensitivity, [5] as well as the three leading theories on more general sensitivity: Differential Susceptibility, [6] [7] Biological Sensitivity to Context, [8] and Sensory processing sensitivity ...
For the full specification of the model, the arrows should be labeled with the transition rates between compartments. Between S and I, the transition rate is assumed to be (/) / = /, where is the total population, is the average number of contacts per person per time, multiplied by the probability of disease transmission in a contact between a susceptible and an infectious subject, and / is ...
Several studies report that differences in response to positive experiences are associated with genetic sensitivity. For example, Keers et al. created a polygenic score for environmental sensitivity based on thousands of gene variants and found that children with higher genetic sensitivity responded more strongly to higher quality of psychological treatment.
In a deterministic model, individuals in the population are assigned to different subgroups or compartments, each representing a specific stage of the epidemic. [17] The transition rates from one class to another are mathematically expressed as derivatives, hence the model is formulated using differential equations.
Gene–environment interaction (or genotype–environment interaction or G×E) is when two different genotypes respond to environmental variation in different ways. A norm of reaction is a graph that shows the relationship between genes and environmental factors when phenotypic differences are continuous. [1]
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