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The periosteum is a membrane that covers the outer surface of all bones, [1] except at the articular surfaces (i.e. the parts within a joint space) of long bones. (At the joints of long bones the bone's outer surface is lined with "articular cartilage", a type of hyaline cartilage .)
Examples of periosteal reactive bone in selected specimens of Triceratops. A periosteal reaction can result from a large number of causes, including injury and chronic irritation due to a medical condition such as hypertrophic osteopathy, bone healing in response to fracture, chronic stress injuries, subperiosteal hematomas, osteomyelitis, and cancer of the bone.
Bone pain originates from both the periosteum and the bone marrow which relay nociceptive signals to the brain creating the sensation of pain. Bone tissue is innervated by both myelinated (A beta and A delta fiber) and unmyelinated sensory neurons. In combination, they can provide an initial burst of pain, initiated by the faster myelinated ...
Mucoperiosteum is a compound structure consisting of mucous membrane and underlying periosteum. It includes epithelium and lamina propria, but attaches directly to the periosteum of underlying bone without the usual submucosa. It consists of loose fatty or glandular tissues; with blood vessels & nerve fibres that supply the mucosa.
The outer surface of a bone is lined by a thin layer of connective tissue that is very similar in morphology and function to endosteum. It is called the periosteum, or the periosteal surface. During bone growth, the width of the bone increases as osteoblasts lay new bone tissue at the periosteum.
Periostitis, also known as periostalgia, is a medical condition caused by inflammation of the periosteum, a layer of connective tissue that surrounds bone. [1] The condition is generally chronic and is marked by tenderness and swelling of the bone and pain.
In the skeletal muscles connected to tendons that pull on bones, the mysia fuses to the periosteum that coats the bone. Contraction of the muscle will transfer to the mysia, then the tendon and the periosteum before causing the bone to move. The mysia also may bind to an aponeurosis or to fascia. [2]
The periosteum, in particular, may create a fibrous band that increases local pressure around the bone, leading to reduced vascularization and bone atrophy. [4] Additionally, thickening of blood vessel walls in the pseudarthrosis area may contribute to the vascularization defect.