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Swan neck deformity has many of possible causes arising from the DIP, PIP, or even the MCP joints. In all cases, there is a stretching of the volar plate at the PIP joint to allow hyperextension, plus some damage to the attachment of the extensor tendon to the base of the distal phalanx that produces a hyperflexed mallet finger.
Chondromalacia patellae (also known as CMP) is an inflammation of the underside of the patella and softening of the cartilage.. The cartilage under the kneecap is a natural shock absorber, and overuse, injury, and many other factors can cause increased deterioration and breakdown of the cartilage.
Truncal ataxia (or trunk ataxia) is a wide-based "drunken sailor" gait characterized by uncertain starts and stops, lateral deviations and unequal steps.It is an instability of the trunk and often seen during sitting. [2]
Additionally, swan-neck deformity can be caused by weakening or tearing of the ligament and tendon on the middle joint of a finger. [10] Other causes of swan-neck deformity include untreated mallet finger, muscle spasticity, physical hand trauma, and many others.
The cerebellum is split into three separate parts: the archicerebellum (controls equilibrium and helps to move the eye, head and neck), midline vermis (helps to move lower body), and lateral hemisphere (control of arms and quick movements). Damage to any part of the cerebellum can cause a disconnect between nerve cells and muscles, causing ...
Dysdiadochokinesia is a feature of cerebellar ataxia and may be the result of lesions to either the cerebellar hemispheres or the frontal lobe (of the cerebrum), it can also be a combination of both. [3]
But those first migraine warning signs can be subtle and unexpected, including symptoms like difficulty concentrating, sleep disturbances, food cravings, neck pain and yawning.
Sensory ataxia is both a symptom and a sign in neurology. It is a form of ataxia (loss of coordination) caused not by cerebellar dysfunction but by loss of sensory input into the control of movement.