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The zygomatic nerve is a branch of the maxillary nerve (itself a branch of the trigeminal nerve (CN V)). It arises in the pterygopalatine fossa and enters the orbit through the inferior orbital fissure before dividing into its two terminal branches: the zygomaticotemporal nerve and zygomaticofacial nerve .
Fractures with displacement require surgery consisting of fracture reduction with miniplates, microplates and screws. Gillie's approach is used for depressed zygomatic fractures. [5] The prognosis of tripod fractures is generally good. In some cases there may be persistent post-surgical facial asymmetry, which can require further treatment. [6]
When zygoma fractures occur, the most typical symptoms are paresthesias in the upper lip, nose, cheek, and lower eyelid, diplopia, and pain.Particular physical characteristics that support zygomatic fracture include globe injury, impaired ocular motility, globe malposition, orbital emphysema, trismus, palpable stepoffs at the inferior or upper lateral edge of the orbit, reduced feeling ...
Oral & Maxillofacial Surgery, ENT surgery, plastic surgery, ophthalmology: Symptoms: Double vision especially when looking up, numbness of the lateral nose skin, the cheek below the eyelid, and the upper lip, Bloody nose, lateral subconjunctival hemorrhage (bright red blood over the sclera (white of the eye)) Causes: Direct trauma to the eye ...
The maxillary sinuses are housed within the maxillary bone, and traumatic injury to these sinuses may cause sinus infections, and changes in eye placement and movement. [4] The infraorbital nerve (a terminal branch of CNV2) courses through the maxillary bone and provides sensation to the central face. Additionally, the maxillary bone contains ...
Treatment is generally based on the extent and location of the injury to structures inside the head. [1] Surgery may be performed to seal a CSF leak that does not stop, to relieve pressure on a cranial nerve or repair injury to a blood vessel. [1] Prophylactic antibiotics do not provide a clinical benefit in preventing meningitis.
Nerve decompressions and resections are the only treatments with a known cure rate. It is a common clinical experience, that even chronic entrapments with longstanding muscle weakness and sensory disturbances sometimes show a very rapid reversibility of some or all of the symptoms after surgical decompression of the nerve. [5]
Often, patients who received surgery following injury or tumor resection suffer extensive nerve damage. This is a serious problem given the importance of facial expressions and speech for communicating in human society. For many who endure such nerve damage, they recover after 12 months; however, others may never fully recover. [36]