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Kingella kingae is a species of Gram-negative facultative anaerobic β-hemolytic coccobacilli. First isolated in 1960 by Elizabeth O. King , it was not recognized as a significant cause of infection in young children until the 1990s, when culture techniques had improved enough for it to be recognized.
Elizabethkingia meningoseptica is a Gram-negative, rod-shaped bacterium widely distributed in nature (e.g. fresh water, salt water, or soil). It may be normally present in fish and frogs; it may be isolated from chronic infectious states, as in the sputum of cystic fibrosis patients.
HACEK is an abbreviation of the initials of the genera of this group of bacteria: Haemophilus, Aggregatibacter (previously Actinobacillus), Cardiobacterium, Eikenella, Kingella. [1] The HACEK organisms are a normal part of the human microbiota , living in the oral - pharyngeal region.
Kingella is a genus of bacteria of the family Neisseriaceae. [1] It belongs to the HACEK group of fastidious Gram-negative bacteria that tend to cause endocarditis . [ 2 ] Kingella kingae is its type species .
Elizabethkingia is a genus of bacterium in the order of Flavobacteriales.It was established in 2005 from a branch in of the genus Chryseobacterium, [1] and named after Elizabeth O. King, the discoverer of the type species. [2]
UniProt is a freely accessible database of protein sequence and functional information, many entries being derived from genome sequencing projects.It contains a large amount of information about the biological function of proteins derived from the research literature.
In the 1960s, King identified a novel bacteria from human respiratory secretions, blood, and bone and joint exudates that was designated Moraxella kingii in her honor shortly after her death. In 1976 it was reassigned to the genus Kingella and given the species name Kingella kingae. [7] [8]
Cross-linking and immunoprecipitation (CLIP, or CLIP-seq) is a method used in molecular biology that combines UV crosslinking with immunoprecipitation in order to identify RNA binding sites of proteins on a transcriptome-wide scale, thereby increasing our understanding of post-transcriptional regulatory networks.