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UVA rays are able to penetrate deeper into the skin than UVB rays, damaging the dermal layer as well as the epidermal. The dermis is the second major layer of the skin and it comprises collagen, elastin, and extrafibrillar matrix which provides structural support to the skin. However, with constant UVA exposure, the size of the dermis layer ...
[45] [failed verification] UVC is the highest-energy, most-dangerous type of ultraviolet radiation, and causes adverse effects that can variously be mutagenic or carcinogenic. [46] Despite the importance of the sun to vitamin D synthesis, it is prudent to limit the exposure of skin to UV radiation from sunlight [47] and from tanning beds. [48]
The sun is, by far, the greatest source of ultraviolet (UV) radiation, which is the culprit that causes most skin cancers—the most common form of cancer in the U.S. According to the Skin Cancer ...
One of the largest promoters of apoptosis is exposure to ultraviolet (UV) light. While UV light is essential to human life it can also cause harm by inducing cancer, immunosuppression, photoaging, inflammation, and cell death. [1] Of the various components of sunlight, ultraviolet radiation B (UVB) (290-320 nm) is considered to be the most harmful.
What it looks like: Actinic keratoses are pre-cancerous changes to the skin caused by sun damage, explains Dr. Zeichner. “The rash feels like sandpaper when you run your fingers over it.”
Artificial UV light sources from tanning units and phototherapy treatment units can also trigger PLE. About three-quarters of patients acquire PLE after UV-A exposure only, one-tenth after UV-B exposure only, and the rest after a combination of UV-A and UV-B exposure. [6] People vary in the amount of sun exposure needed to trigger the rash. [15]
Excessive UV radiation is the leading cause of (primarily) non-malignant skin tumors, [1] [2] which in extreme cases can be life-threatening. Sunburn is an inflammatory response in the tissue triggered by direct DNA damage by UV radiation. When the cells' DNA is overly damaged by UV radiation, type I cell-death is triggered and the tissue is ...
Phytophotodermatitis, also known as berloque dermatitis, [1] [2] [3] margarita photodermatitis, [4] [5] lime disease [6] or lime phytodermatitis [6] is a cutaneous phototoxic inflammatory reaction resulting from contact with a light-sensitizing botanical agent (such as lime juice) followed by exposure to ultraviolet A (UV-A) light (from the sun, for instance).