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Idiopathic guttate hypomelanosis is characterised by multiple small whitish flat spots. [1] They are typically irregular, well defined and frequently appear on the arms, legs, and faces of older people. [1] It occurs in up to 80% of people over 70 years old. [2] Females may notice it at a younger age than males. [3]
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Skin pigmentation is a frequent disorder that has a number of potential causes. Genetics, sun exposure, and some drugs are the three main factors that contribute to skin pigmentation.
Generally, diseases outlined within the ICD-10 codes L80-L81 within Chapter XII: Diseases of the skin and subcutaneous tissue should be included in this category. Disturbances of human pigmentation, either loss or reduction, may be related to loss of melanocytes or the inability of melanocytes to produce melanin or transport melanosomes correctly.
Linear and whorled nevoid hypermelanosis (also known as "Linear nevoid hyperpigmentation," "Progressive cribriform and zosteriform hyperpigmentation," "Reticulate and zosteriform hyperpigmentation," "Reticulate hyperpigmentation of Iijima and Naito and Uyeno," "Zebra-like hyperpigmentation in whorls and streaks," and "Zebra-line hyperpigmentation" [1]) is a disorder of pigmentation that ...
Nevus of Ota (congenital melanosis bulbi, melanosis bulborum and aberrant dermal melanocytosis, nevus fuscoceruleus ophthalmomaxillaris, oculodermal melanocytosis, oculomucodermal melanocytosis) Nevus spilus (speckled lentiginous nevus, zosteriform lentiginous nevus) Partial unilateral lentiginosis (segmental lentiginosis) Peutz–Jeghers syndrome
Melanosis is a form of hyperpigmentation associated with increased melanin. [1] It can also refer to: Melanism; Ocular melanosis; Smoker's melanosis; Oral melanosis;
Piebaldism is an autosomal dominant [4] hereditary condition, which tends to produce high rates of inheritance and long chains of generational transmission. All who inherit the gene have at some time in life evidence of piebald hypopigmentation of the hair or skin, most likely both.