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Both macular telangiectasia and age-related macular degeneration (AMD) lead to photoreceptor atrophy and loss of central vision. The two diseases may be distinguished by symptoms, clinical features, and pathogenesis. The natural history of macular telangiectasia suggests a slowly progressive disorder.
Telangiectasia macularis eruptiva perstans (TMEP) is persistent, pigmented, asymptomatic eruption of macules usually less than 0.5 cm in diameter with a slightly reddish-brown tinge. [ 1 ] : 616 [ 2 ]
Macular scarring is formation of the fibrous tissue in place of the normal retinal tissue on the macular area of the retina which provides the sharpest vision in the eyes. It is usually a result of an inflammatory or infectious process. [1] Other etiologies include macular pucker (macular detachment), macular hole, and age-related macular ...
Epiretinal membrane or macular pucker is a disease of the eye in response to changes in the vitreous humor or more rarely, diabetes.Sometimes, as a result of immune system response to protect the retina, cells converge in the macular area as the vitreous ages and pulls away in posterior vitreous detachment (PVD).
Ophthalmology (/ ˌ ɒ f θ æ l ˈ m ɒ l ə dʒ i /, OFF-thal-MOL-ə-jee) [1] is a clinical and surgical specialty within medicine that deals with the diagnosis and treatment of eye disorders. [2] A former term is oculism. An ophthalmologist is a physician who undergoes subspecialty training in medical and surgical eye care. [3]
Other signs may include telangiectasia on the anterior eyelid, collarettes encircling the lash base, and corneal changes. [2] Seborrheic blepharitis is distinguished by less erythema, edema, and telangiectasia of the eyelid margins. Posterior blepharitis and Meibomian gland dysfunction are frequently associated with rosacea and can be seen ...
Polypoidal choroidal vasculopathy (PCV) is an eye disease primarily affecting the choroid.It may cause sudden blurring of vision or a scotoma in the central field of vision.
The etiology of multiple evanescent white dot syndrome is currently unknown but two potential origins have been postulated. Gass, et. al. suggests a virus invades retinal photoreceptors through cell-to-cell transmission via either the ora serrata or optic disc margin. [3]