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Age: Primary herpetic gingivostomatitis is common in children from 6 months to 5 years old. This virus is also common in young adults aged around 20–25. [5] Immune system: The prevalence and severity of the disease is dependent on the host's immune response and the virulence of the virus.
High-risk children typically need more intensive treatment. This may include early restorative work to repair and address any existing decay to prevent further deterioration of the teeth. Since ECC affects children under the age of 5 years, dental treatments under general anesthesia may be necessary in select cases. [14]
The term "full width gingivitis" usually refers to the oral lesions of orofacial granulomatosis however. [4] The color is another dissimilarity between typical marginal gingivitis and desquamative gingivitis, in the latter it is dusky red. [3] Plasma cell gingivitis is another form of gingivitis which affects both the attached and free gingiva. [1]
In developed countries, necrotizing gingivitis occurs mostly in young adults with predisposing factors such as psychological stress, sleep deprivation, poor oral hygiene, smoking, immunosuppression and/or malnutrition. In developing countries, necrotizing gingivitis occurs mostly in malnourished children.
Plasma cell gingivitis in a 10-year-old child. Histologically verified. Plasma cell gingivitis appears as mild gingival enlargement and may extend from the free marginal gingiva on to the attached gingiva. [6] Sometimes it is blended with a marginal, plaque induced gingivitis, or it does not involve the free marginal gingiva. It may also be ...
Hand, foot and mouth disease most commonly occurs in children under the age of 10 [4] [19] and more often under the age of 5, but it can also affect adults with varying symptoms. [20] It tends to occur in outbreaks during the spring, summer, and autumn seasons. [6] This is believed to be due to heat and humidity improving spread. [22]
While in some individuals gingivitis never progresses to periodontitis, [14] periodontitis is always preceded by gingivitis. [15] In 1976, Page & Schroeder [16] introduced an innovative new analysis of periodontal disease based on histopathologic and ultrastructural features of the diseased gingival tissue.
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