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Hemifacial spasm (HFS) is a rare neuromuscular disease characterized by irregular, involuntary muscle contractions on one side (hemi-) of the face (-facial). [1] The facial muscles are controlled by the facial nerve (seventh cranial nerve), which originates at the brainstem and exits the skull below the ear where it separates into five main branches.
Microvascular decompression (MVD), also known as the Jannetta procedure, [1] is a neurosurgical procedure used to treat trigeminal neuralgia (along with other cranial nerve neuralgias), a pain syndrome characterized by severe episodes of intense facial pain, and hemifacial spasm.
The Chvostek sign is the abnormal twitching of muscles that are activated (innervated) by the facial nerve (also known as Cranial Nerve Seven, or CNVII). [1] When the facial nerve is tapped in front of the ear, the facial muscles on the same side of the face will contract sporadically (called ipsilateral facial spasm). The muscles that control ...
The facial nerve, also known as the seventh cranial nerve, cranial nerve VII, or simply CN VII, is a cranial nerve that emerges from the pons of the brainstem, controls the muscles of facial expression, and functions in the conveyance of taste sensations from the anterior two-thirds of the tongue.
Tumour of facial nerve like schwannomas and perineuromas. Other tumours that can compress facial nerve along its course like congenital cholesteatomas, hemangiomas, acoustic neuromas, parotid gland neoplasms, or metastases of other tumors. Other causes like viral, bacterial or fungal infections like chicken pox, streptococcal infection or ...
Bilateral facial nerve paralysis may occur in Guillain–Barré syndrome, an autoimmune condition of the peripheral nervous system. [6] Moebius syndrome is a bilateral facial paralysis resulting from the underdevelopment of the VII cranial nerve (facial nerve), which is present at birth. The VI cranial nerve, which controls lateral eye movement ...
The zygomatic branches of the facial nerve (malar branches) are nerves of the face. They run across the zygomatic bone to the lateral angle of the orbit . Here, they supply the orbicularis oculi muscle , and join with filaments from the lacrimal nerve and the zygomaticofacial branch of the maxillary nerve (CN V 2 ).
Møller found evidence that the spasm was created by the facial motor nucleus probably through the process of activation of neuroplasticity and not by the pathologies of the facial nerve where it was in contact with a blood vessel. [17] These studies also improved the outcome of operations for hemifacial spasm.