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  2. Factor V Leiden - Wikipedia

    en.wikipedia.org/wiki/Factor_V_Leiden

    Factor V Leiden (rs6025 or F5 p.R506Q [1]) is a variant (mutated form) of human factor V (one of several substances that helps blood clot), which causes an increase ...

  3. Factor V - Wikipedia

    en.wikipedia.org/wiki/Factor_V

    Coagulation factor V (Factor V), also less commonly known as proaccelerin or labile factor, is a protein involved in coagulation, encoded, in humans, by F5 gene. [5] In contrast to most other coagulation factors, it is not enzymatically active but functions as a cofactor . [ 5 ]

  4. Activated protein C resistance test - Wikipedia

    en.wikipedia.org/wiki/Activated_protein_C...

    [1] [5] This ratio is inversely related to the degree of APC resistance. [7] The ETP-based APC resistance test involves the addition of APC to a thrombin generation assay (TGA). [5] This results in an inhibition of thrombin generation as measured by reduction of the endogenous thrombin potential (ETP; area under the thrombin generation curve). [5]

  5. Thrombophilia - Wikipedia

    en.wikipedia.org/wiki/Thrombophilia

    The minor ("type 2") thrombophilias are much more common. Factor V Leiden is present in 5% of the population of Northern European descent, but much rarer in those of Asian or African extraction. In people with thrombosis, 10% have factor V Leiden. In those who are referred for thrombophilia testing, 30–50% have the defect.

  6. Activated protein C resistance - Wikipedia

    en.wikipedia.org/wiki/Activated_protein_C_resistance

    APC resistance is the inability of protein C to cleave Factor Va and/or Factor VIIIa, which allows for longer duration of thrombin generation and may lead to a hypercoagulable state. This may be hereditary or acquired. [4] The best known and most common hereditary form is Factor V Leiden, which is responsible for more than 95% of cases. [5]

  7. Splenic infarction - Wikipedia

    en.wikipedia.org/wiki/Splenic_infarction

    Splenic infarct seen on CT Healed splenic infarct. Several factors may increase the tendency for clot formation, such as specific infections (such as infectious mononucleosis, [9] [dubious – discuss] [better source needed] cytomegalovirus infection, [10] malaria, [11] or babesiosis [12]), inherited clotting disorders (thrombophilia, such as Factor V Leiden, antiphospholipid syndrome ...

  8. Thromboembolism is a well-described complication of IBD, with a clinical incidence of up to 6% and a three-fold higher risk of disease, [31] [32] and the Factor V Leiden mutation further increases the risk of venous thrombosis. [33] Recent studies describe the co-occurrence between coeliac disease, in which IBD is common in venous thrombosis ...

  9. Factor V Leiden mutation - Wikipedia

    en.wikipedia.org/?title=Factor_V_Leiden_mutation&...

    This page was last edited on 24 November 2003, at 09:57 (UTC).; Text is available under the Creative Commons Attribution-ShareAlike 4.0 License; additional terms may apply.