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This is a list of Superfund sites in Maine designated under the Comprehensive Environmental Response, Compensation, and Liability Act (CERCLA) environmental law. The CERCLA federal law of 1980 authorized the United States Environmental Protection Agency (EPA) to create a list of polluted locations requiring a long-term response to clean up hazardous material contaminations. [1]
Toxicity characteristic leaching procedure (TCLP) is a soil sample extraction method for chemical analysis employed as an analytical method to simulate leaching through a landfill. The testing methodology is used to determine if a waste is characteristically hazardous, i.e., classified as one of the "D" listed wastes by the U.S. Environmental ...
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Sep. 1—Contaminated waters could hinder beach adventures from York to Camden this Labor Day weekend. The Maine Department of Environmental Protection has issued contamination warnings for five ...
The U.S. Environmental Protection Agency declared the base a Superfund Site in 1987 for chemical contamination after open-air detonation of ordnance. From 2007 until 2015, the Navy assessed and removed ordnance from the area, spending $3–4 million on site clean-up.
Freedom Industries was required to test the rain runoff every quarter, and send the results to the DEP. [8] Under the federal Emergency Planning and Community Right-to-Know Act, Freedom Industries filed its "Tier Two" form in February 2013, which contained detailed information about each chemical it was storing at its Charleston facility. [10]
To ensure consistency and repeatability, the methods use in the chemical analysis of water samples are often agreed and published at a national or state level. By convention these are often referred to as "Blue book". [4] [5] Certain analyses are performed in-field (e.g. pH, specific conductance) while others involve sampling and laboratory ...
Little is known about the chronic toxicity of diethyl phthalate, but existing information suggests only a low toxic potential. [6] Studies suggest that some phthalates affect male reproductive development via inhibition of androgen biosynthesis.