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Both macular telangiectasia and age-related macular degeneration (AMD) lead to photoreceptor atrophy and loss of central vision. The two diseases may be distinguished by symptoms, clinical features, and pathogenesis. The natural history of macular telangiectasia suggests a slowly progressive disorder.
The presence of systemic involvement or clinical symptoms determines how individuals with TMEP are treated. There is no gold standard medication for the treatment of TMEP, thus it is critical to identify and avoid conditions that trigger mast cell destruction, such as sunshine, severe temperatures, alcohol , and narcotics .
Polypoidal choroidal vasculopathy (PCV) is an eye disease primarily affecting the choroid.It may cause sudden blurring of vision or a scotoma in the central field of vision.
The onset of ocular symptoms are usually preceded by episode of viral or flu-like symptoms such as fever, cough or sore throat (however this is not always the case). Patients can typically present erythema nodosum , livedo reticularis , bilateral uveitis , and sudden onset of marked visual loss associated with the appearance of multiple lesions ...
Macular scarring is formation of the fibrous tissue in place of the normal retinal tissue on the macular area of the retina which provides the sharpest vision in the eyes. It is usually a result of an inflammatory or infectious process. [1] Other etiologies include macular pucker (macular detachment), macular hole, and age-related macular ...
Many ophthalmic imaging techniques are used in detecting hypotony maculopathy. Indocyanine green angiography or fluorescein angiography can help in early detection of choroidal disturbances and choroidal folds.
Patients with branch retinal vein occlusion usually have a sudden onset of blurred vision or a central visual field defect. The eye examination findings of acute branch retinal vein occlusion include superficial hemorrhages, retinal edema, and often cotton-wool spots in a sector of retina drained by the affected vein.
The etiology of multiple evanescent white dot syndrome is currently unknown but two potential origins have been postulated. Gass, et. al. suggests a virus invades retinal photoreceptors through cell-to-cell transmission via either the ora serrata or optic disc margin. [3]