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Atrial fibrillation with rapid ventricular response; Atrial flutter with rapid ventricular response (Without rapid ventricular response, fibrillation and flutter are usually not classified as SVT) Atrioventricular origin: [22] AV nodal reentrant tachycardia (AVNRT) or junctional reciprocating tachycardia (JRT)
Bypassing the AV node, the atrial rate of 300 beats/minute leads to a ventricular rate of 300 beats/minute (1:1 conduction). Even if the ventricles are able to sustain a cardiac output at such a high rate, 1:1 flutter with time may degenerate into ventricular fibrillation, causing hemodynamic collapse and death. [9]
Atrial fibrillation (AF, AFib or A-fib) is an abnormal heart rhythm (arrhythmia) characterized by rapid and irregular beating of the atrial chambers of the heart. [11] [12] It often begins as short periods of abnormal beating, which become longer or continuous over time. [4]
[29] [30] [31] The distinction is that tachycardia be reserved for the rapid heart rate itself, regardless of cause, physiologic or pathologic (that is, from healthy response to exercise or from cardiac arrhythmia), and that tachyarrhythmia be reserved for the pathologic form (that is, an arrhythmia of the rapid rate type).
Wolff–Parkinson–White syndrome (WPWS) is a disorder due to a specific type of problem with the electrical system of the heart involving an accessory pathway able to conduct electrical current between the atria and the ventricles, thus bypassing the atrioventricular node.
Ashman beats are described as wide complex QRS complexes that follow a short R-R interval preceded by a long R-R interval. [3] This short QRS complex typically has a right bundle branch block morphology and represents an aberrantly conducted complex that originates above the AV node, rather than a complex that originates in either the right or left ventricle.
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Atrial tachycardia is a type of heart rhythm problem in which the heart's electrical impulse comes from an ectopic pacemaker (that is, an abnormally located cardiac pacemaker) in the upper chambers of the heart, rather than from the sinoatrial node, the normal origin of the heart's electrical activity.