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Atrial fibrillation is associated with an increased risk of heart failure, dementia, and stroke. [3] [12] It is a type of supraventricular tachycardia. [14] Atrial fibrillation frequently results from bursts of tachycardia that originate in muscle bundles extending from the atrium to the pulmonary veins. [15]
Ashman beats are described as wide complex QRS complexes that follow a short R-R interval preceded by a long R-R interval. [3] This short QRS complex typically has a right bundle branch block morphology and represents an aberrantly conducted complex that originates above the AV node, rather than a complex that originates in either the right or left ventricle.
Atrial fibrillation: Irregular impulses reaching AV node, only some being transmitted. The following types of supraventricular tachycardias are more precisely classified by their specific site of origin. While each belongs to the broad classification of SVT, the specific term/diagnosis is preferred when possible: Sinoatrial origin: [23]
The types of SVT associated with TIC include atrial fibrillation, atrial flutter, incessant atrial tachycardia, permanent junctional reciprocating tachycardia, atrioventricular reciprocating tachycardia, and atrioventricular nodal reentry tachycardia. [1] Atrial fibrillation is the most common and well-studied etiology of TIC. [1] [5]
Catheter ablation is a procedure that uses radio-frequency energy or other sources to terminate or modify a faulty electrical pathway from sections of the heart of those who are prone to developing cardiac arrhythmias such as atrial fibrillation, atrial flutter and Wolff-Parkinson-White syndrome.
Rheumatic heart disease, heart failure, atrial fibrillation, infection of the valves [1] Usual onset: 2–4 weeks after a streptococcal throat infection, age 5–14 years [2] Causes: Autoimmune disease triggered by pathogenic strains of Streptococcus [1] Risk factors: Genetic factors, malnutrition and poverty. [1] Diagnostic method
Atrial tachycardia is a type of heart rhythm problem in which the heart's electrical impulse comes from an ectopic pacemaker (that is, an abnormally located cardiac pacemaker) in the upper chambers of the heart, rather than from the sinoatrial node, the normal origin of the heart's electrical activity.
However, atrial flutter is more resistant to correction with such medications than atrial fibrillation. [1] For example, although the class III antiarrhythmic agent ibutilide is an effective treatment for atrial flutter, rates of recurrence after treatment are quite high (70–90%). [ 1 ]