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Crohn's disease is influenced by genetic, environmental, and immunological factors. Smoking is a major modifiable risk factor, especially in Western countries, where it doubles the likelihood of developing the disease. Dietary shifts from high-fiber to processed foods may reduce microbiota diversity and increase risk, while high-fiber diets can ...
As of 2019, about 4.9 million people globally were living with an inflammatory bowel disease (IBD) such as Crohn’s disease. Scientists are still unclear as to the exact cause of Crohn’s ...
In Crohn's disease, surgery involves removing the worst inflamed segments of the intestine and connecting the healthy regions, but unfortunately, it does not cure Crohn's or eliminate the disease. At some point after the first surgery, Crohn's disease can recur in the healthy parts of the intestine, usually at the resection site. [76] (For ...
Crohn’s disease is a type of inflammatory bowel disease (IBD). It can cause inflammation to any part or layer of your bowel, although most often, it affects the end of your small bowel to the ...
He has explored the aspects of genetic architecture, genetic variability, and population genetics whereas his main disease focus has been inflammatory bowel diseases, including Crohn's disease and ulcerative colitis. Other research foci have been the diseases psoriasis and primary sclerosing cholangitis. [2] [10]
The anti-TNF-α monoclonal antibody infliximab is a major biological therapy for inflammatory bowel diseaseBiological therapy, the use of medications called biopharmaceuticals or biologics that are tailored to specifically target an immune or genetic mediator of disease, plays a major role in the treatment of inflammatory bowel disease. [1]
Genetic variants associated with Alzheimer disease, deep venous thrombosis, Crohn disease, and type 2 diabetes appear to adhere to this model. [20] However, the generality of the model has not yet been established and, in some cases, is in doubt.
There is no association between genetic markers for Crohn's disease and NOD2 protein (also known as CARD15) or antibodies to mycoprotein antigen (IgA or IgG), indicating heterogeneous causes for Crohn's disease. [23] Experimental studies show that chemically induced colitis promotes opportunistic human fungal pathogen Candida albicans colonization.
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