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Dimethylamphetamine (Metrotonin), also known as dimetamfetamine , dimephenopan and N,N-dimethylamphetamine, is a stimulant drug of the phenethylamine and amphetamine chemical classes. Dimethylamphetamine has weaker stimulant effects than amphetamine or methamphetamine and is considerably less addictive [ 1 ] and less neurotoxic compared to ...
There is also evidence suggesting that N,N-DMPEA acts as a TAAR1 agonist in humans, [5] and as a 5-HT1A ligand in rats. Some less conclusive research also indicated that it had interaction with MAO-B, most likely as an enzyme substrate and not an inhibitor. [6] N,N-DMPEA is a positional isomer of methamphetamine. Instead of the methyl group ...
key:nfrbjbfyunqiqp-uhfffaoysa-n N , N -Dimethyl-4-methylthioamphetamine ( DMMTA or N , N -dimethyl-4-MTA ; code name PAL-1062 ), also known as 4-methylthio- N , N -dimethylamphetamine ( 4-MTDMA ), is a monoamine releasing agent (MRA) of the amphetamine family related to 4-methylthioamphetamine (4-MTA) and 4-methylthiomethamphetamine (4-MTMA or ...
N,N-Dimethylethylamine (DMEA), sometimes referred to as dimethylethylamine, is an organic compound with formula (CH 3) 2 NC 2 H 5.It is an industrial chemical that is mainly used in foundries as a catalyst for epoxy resins and polyurethane as well as sand core production.
N,N′-Di-2-butyl-1,4-phenylenediamine is an aromatic amine used industrially as an antioxidant to prevent degradation of turbine oils, transformer oils, hydraulic fluids, lubricants, waxes, and greases. It is particularly effective for hydrocarbon products produced by cracking or pyrolysis, which are characterized by high alkene content.
N-Methylphenethylamine (NMPEA) is a naturally occurring trace amine neuromodulator in humans that is derived from the trace amine, phenethylamine (PEA). [2] [3] It has been detected in human urine (<1 μg over 24 hours) [4] and is produced by phenylethanolamine N-methyltransferase with phenethylamine as a substrate, which significantly increases PEA's effects.
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The blood level decline of the parent drug was biphasic with the short half-life ranging from 0.5–0.7 hours and the terminal half-life from 8 to 26.5 hours (mean 17.25 hours). [citation needed] It is the N-methylated analogue of nitrazepam (Mogadon, Alodorm), to which it is partially metabolized.