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Viral cardiomyopathy occurs when viral infections cause myocarditis with a resulting thickening of the myocardium and dilation of the ventricles. These viruses include Coxsackie B and adenovirus, echoviruses, influenza H1N1, Epstein–Barr virus, rubella (German measles virus), varicella (chickenpox virus), mumps, measles, parvoviruses, yellow fever, dengue fever, polio, rabies, and the ...
This presentation is known as pleurodynia or Bornholm disease in many areas. Patients with chest pain should see a doctor immediately—in some cases, viruses in the Coxsackie B family progress to myocarditis or pericarditis, which can result in permanent heart damage or death. Coxsackie B virus infection may also induce aseptic meningitis.
The amount of virally infected cardiomyocytes varies in different stages of the disease. In a mouse model, at the acute stage (7 days after infection with coxsackievirus B3) approximately 10% of the myocytes are infected and could affect overall cardiac function. In chronic murine infection, the percentage of infected cardiomyocytes are much lower.
Cardiovirus A is composed of only one serotype, encephalomyocarditis virus (EMCV). Cardiovirus B consists of four viruses that are most probably serologically distinct. These are Theiler's Murine encephalomyelitis virus (TMEV), Vilyuisk human encephalomyelitis virus (VHEV), a Theiler-like rat virus (TRV) (which has yet to be named) and Saffold ...
But heart disease is multifactorial,” Dr. Tadwalkar says. He stresses that genetics plays a big role, along with diet, exercise routine, and other environmental elements like air quality .
Myocarditis can be seen during COVID-19, the disease caused by the SARS-CoV-2 virus; [60] with the myocarditis being associated with a spectrum of severities from asymptomatic to fulminant. The symptoms for myocarditis following a COVID-19 infection can present as chest pain, shortness of breath, fatigue, and irregular heartbeats which can make ...
The coronavirus can damage the heart, according to a major new study which found abnormalities in the heart function of more than half of patients.
The cytoplasmic tail of CAR contains the amino acids G S I V, which is characterized as a class 1 PDZ-binding motif for interacting with proteins containing PDZ domains. [11] The protein is found to be expressed in various regions of the body including the heart, brain, and, more generally, epithelial and endothelial cells. Moreover, CAR ...