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Endometrial hyperplasia is a condition of excessive proliferation of the cells of the endometrium, or inner lining of the uterus.. Most cases of endometrial hyperplasia result from high levels of estrogens, combined with insufficient levels of the progesterone-like hormones which ordinarily counteract estrogen's proliferative effects on this tissue.
An atypical hyperplasia is one with visible abnormalities in the nuclei. Pre-cancerous endometrial hyperplasias are also referred to as endometrial intraepithelial neoplasia. [41] Mutations in the KRAS gene can cause endometrial hyperplasia and therefore Type I endometrial cancer. [37] Endometrial hyperplasia typically occurs after the age of ...
Megestrol acetate was reported to be effective in the treatment of endometrial hyperplasia in the mid-1960s. [6] It first started to be studied as a treatment for endometrial cancer in 1967, with findings published in 1973. [6] [156] [157] Megestrol acetate was reportedly introduced for the treatment of endometrial cancer in the United States ...
Hyperplasia of the breast – "Hyperplastic" lesions of the breast include usual ductal hyperplasia, a focal expansion of the number of cells in a terminal breast duct, and atypical ductal hyperplasia, in which a more abnormal pattern of growth is seen, and which is associated with an increased risk of developing breast cancer. [2] Intimal ...
HDE and/or pseudopregnancy have been used in clinical medicine for the following indications: Estrogen receptor-positive breast cancer in women [1]; As a means of androgen deprivation therapy for prostate cancer and benign prostatic hyperplasia in men [7] [8] [10]
Atypical hyperplasia is a high-risk premalignant lesion of the breast. It is believed that atypical ductal hyperplasia (ADH) is a direct precursor for low-grade mammary ductal carcinoma , whereas atypical lobular hyperplasia (ALH) serves as a risk indicator.
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