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  2. Fructolysis - Wikipedia

    en.wikipedia.org/wiki/Fructolysis

    Hexokinase IV (Glucokinase), also occurs in the liver and would be capable of phosphorylating fructose to fructose 6-phosphate (an intermediate in the gluconeogenic pathway); however, it has a relatively high Km (12 mM) for fructose and, therefore, essentially all of the fructose is converted to fructose-1-phosphate in the human liver.

  3. Metabolic dysfunction–associated steatotic liver disease

    en.wikipedia.org/wiki/Metabolic_dysfunction...

    [35] [58] Furthermore, high fructose consumption promotes fat accumulation in the liver by stimulating de novo lipogenesis in the liver and reducing the beta-oxidation of fat. [20] Unlike the sugar glucose, the enzyme fructokinase rapidly metabolizes fructose. This leads to a decreased level of intracellular adenosine triphosphate (ATP). [20]

  4. The 4 Worst Drinks if You're Trying to Lose Visceral Fat ...

    www.aol.com/4-worst-drinks-youre-trying...

    Research has also linked high fructose consumption, a type of sugar commonly found in sodas, to increased visceral fat levels. ... beverages for this reason. For example, according to the USDA, a ...

  5. 30 Day High-Fiber Meal Plan to Help You Lose Visceral Fat ...

    www.aol.com/30-day-high-fiber-meal-134500888.html

    Research links a high-fiber diet with a reduced level of visceral fat and less abdominal weight gain with age. We also include an average of 87 grams of protein per day. Like fiber, protein plays ...

  6. Lipogenesis - Wikipedia

    en.wikipedia.org/wiki/Lipogenesis

    In biochemistry, lipogenesis is the conversion of fatty acids and glycerol into fats, or a metabolic process through which acetyl-CoA is converted to triglyceride for storage in fat. [1] Lipogenesis encompasses both fatty acid and triglyceride synthesis , with the latter being the process by which fatty acids are esterified to glycerol before ...

  7. De novo synthesis - Wikipedia

    en.wikipedia.org/wiki/De_novo_synthesis

    The reduction of adipose tissue de novo lipogenesis, and the increase in liver de novo lipogenesis due to obesity and insulin resistance leads to fatty liver disease. Fructose consumption (in contrast to glucose) activates both SREBP-1c and ChREBP in an insulin independent manner. [8] Although glucose can be converted into glycogen in the liver ...