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Dispersin B is produced by Aggregatibacter actinomycetemcomitans, a Gram-negative oral bacterium, when it needs to detach and disperse adherent bacterial cells. [4] A. actinomycetemcomitans forms asymmetric biofilm lobed colonies that release single cells or small clusters of bacterial cells, which can attach to nearby surfaces, form new colonies, and enable the biofilm to spread.
Dispersal of cells from the biofilm colony is an essential stage of the biofilm life cycle. Dispersal enables biofilms to spread and colonize new surfaces. Enzymes that degrade the biofilm extracellular matrix, such as dispersin B and deoxyribonuclease, may contribute to biofilm dispersal.
Aggregatibacter actinomycetemcomitans is a Gram-negative, facultative anaerobe, nonmotile bacterium that is often found in association with localized aggressive periodontitis, a severe infection of the periodontium.
In P. aeruginosa infections, quorum sensing is critical for biofilm formation and pathogenicity. [22] P. aeruginosa contains two pairs of LuxI/LuxR homologs, LasI/LasR and RhlI, RhlR. [23] [24] LasI and RhlI are synthase enzymes that catalyze the synthesis of N-(3-oxododecanoyl)-homoserine lactone and N-(butyryl)-homoserine lactone, respectively.
Biofilms can consist of a multitude of bacteria, fungi, and algae which are able to absorb, immobilize, and degrade many common pollutants found in wastewater.By harnessing a natural phenomenon, biofilm-mediated remediation is an environmentally friendly method for environmental cleanup. [3]
The C2DA inhibit methicillin resistant staphylococcus biofilm, but don't eliminate it. The mechanism of the biofilm inhibition by these molecules is still unknown. C2D is a medium of fatty acid chain that effect on staphylococcus aureus biofilm and dispersion of these biofilm. Pseudomonas aeruginosa is the main source for these molecules. [15]
B. subtilis has gained interest for its probiotic properties due to its biofilm which allows it to effectively maintain a favorable microenvironment in the gastrointestinal tract. In order to survive the passage through the upper gastrointestinal tract, B. subtilis produces an extracellular matrix that protects it from stressful environments ...
In 1996 the national profile of the CBE and biofilm research was on the rise. Numerous scientific and mass media publications began to address biofilm technology in earnest. An article in the September 1996 issue of Science, entitled "Biofilms Invade Microbiology" featured the work and history of the Center for Biofilm Engineering. [4]
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