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Several models have been put forward to explain the link between altered brain function and schizophrenia. [27] The prevailing model of schizophrenia is that of a neurodevelopmental disorder, and the underlying changes that occur before symptoms become evident are seen as arising from the interaction between genes and the environment. [126]
Recent studies have used fMRI to explore specific brain networks, such as the salience network and default mode network, to understand their roles in schizophrenia-related symptoms. Alterations in these networks may affect self-referential thoughts and responses to external stimuli, potentially contributing to symptoms like hallucinations and ...
The causes of schizophrenia that underlie the development of schizophrenia, a psychiatric disorder, are complex and not clearly understood.A number of hypotheses including the dopamine hypothesis, and the glutamate hypothesis have been put forward in an attempt to explain the link between altered brain function and the symptoms and development of schizophrenia.
These rapid evolutionary changes, in some cases, may impede normal development within the social brain. [14] [15] As schizophrenia is foremost a disorder of the consciousness, it has been suggested that schizophrenia exists as an unwanted byproduct of the evolution of the prefrontal cortex and other brain regions constituting the social brain. [13]
Ketamine produces more similar symptoms (hallucinations, withdrawal) without observed permanent effects (other than ketamine tolerance). Both arylcyclohexamines have some(uM) affinity to D2 and as triple reuptake inhibitors. PCP is representative symptomatically, but does appear to cause brain structure changes seen in schizophrenia. [22]
People with schizophrenia are at a higher than average risk of physical ill health, and earlier death than the general population. [1] [2] [3] The fatal conditions include cardiovascular, respiratory and metabolic disorders. [4]
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