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Toxoplasmosis is a parasitic disease caused by Toxoplasma gondii, an apicomplexan. [3] Infections with toxoplasmosis are associated with a variety of neuropsychiatric and behavioral conditions. [8] Occasionally, people may have a few weeks or months of mild, flu-like illness such as muscle aches and tender lymph nodes. [1]
Dividing T. gondii parasites. Toxoplasma gondii (/ ˈ t ɒ k s ə ˌ p l æ z m ə ˈ ɡ ɒ n d i. aɪ,-iː /) is a species of parasitic alveolate that causes toxoplasmosis. [3] Found worldwide, T. gondii is capable of infecting virtually all warm-blooded animals, [4]: 1 but felids are the only known definitive hosts in which the parasite may undergo sexual reproduction.
The effects of parasitic worms, or helminths, on the immune system is a recently emerging topic of study among immunologists and other biologists. Experiments have involved a wide range of parasites, diseases, and hosts. The effects on humans have been of special interest.
Congenital toxoplasmosis may lead to hydrocephalus, seizures, lymphadenopathy, hepatosplenomegaly, rash, and fever. However, retinochoroiditis is the most common manifestation, occurring in 3/4 of cases. In congenital toxoplasmosis, the disease is bilateral in 65–85% of cases and involves the macula in 58%.
Main article: Human parasite Endoparasites Protozoan organisms Common name of organism or disease Latin name (sorted) Body parts affected Diagnostic specimen Prevalence Source/Transmission (Reservoir/Vector) Granulomatous amoebic encephalitis and Acanthamoeba keratitis (eye infection) Acanthamoeba spp. eye, brain, skin culture worldwide contact lenses cleaned with contaminated tap water ...
Toxoplasmosis life cycle between humans and animals. Balantidium coli (Balantidiasis) is an example of a member of the phylum Ciliophora. Balantidiasis is the only ciliate known to be capable of infecting humans, and swine are the primary reservoir host. [27] Balantidiasis is opportunistic and rare in Western countries. [28]
Viruses from the family Baculoviridae induce in their hosts changes to both feeding behavior and environment selection. They infect moth and butterfly caterpillars, which, sometime following infection, begin to eat incessantly, providing nutrients for the virus's replication.
The PV prevents the acidification of the compartment, the mechanism by which the lysosomes of the host cell would normally destroy an invading parasite. [1] Parasites that form a parasitophorous vacuole as part of their infection process include Plasmodium falciparum , which causes malaria and Toxoplasma gondii , which causes toxoplasmosis .