Search results
Results From The WOW.Com Content Network
Neurotoxicity is a form of toxicity in which a biological, chemical, or physical agent produces an adverse effect on the structure or function of the central and/or peripheral nervous system. [1]
NeuroToxicology is a peer-reviewed scientific journal covering research on the toxicology of the nervous system. It was established in 1979 and originally published by Intox Press, until it was acquired by Elsevier in 2001. The editor-in-chief is Joan Marie Cranmer (University of Arkansas for Medical Sciences).
Though clinical neurotoxicology is largely a burgeoning field, extensive inroads have been made in the identification of many environmental neurotoxins leading to the classification of 750 to 1000 known potentially neurotoxic compounds. [21]
Neurotoxicology and Teratology is a bimonthly peer-reviewed scientific journal covering research on the toxicological effects of chemical and physical agents on the nervous system. It was established in 1979 as Neurobehavioral Toxicology , was renamed to Neurobehavioral Toxicology and Teratology in 1981, and obtained its current title in 1987.
Olney's lesions, also known as NMDA receptor antagonist neurotoxicity (NAT), is a form of brain damage consisting of selective death of neurons but not glia, observed in restricted brain regions of rats and certain other animal models exposed to large quantities of psychoactive drugs that inhibit the normal operation of the neuronal NMDA receptor.
This page was last edited on 30 July 2006, at 05:36 (UTC).; Text is available under the
Toshio Narahashi (January 30, 1927 – April 21, 2013) was an internationally known pharmacologist. He was the John Evans Professor of Pharmacology and former chair of the Department of Pharmacology at Northwestern University Feinberg School of Medicine, where he served on the faculty from 1977 to 2013.
Neurotoxicology [ edit ] Darryl B. Hood's work in the research of polycyclic aromatic hydrocarbons , as well as benzo(a)pyrene , (B(a)P) has shown that utero exposure to B(a)P results in a diminished expression of specific NMDA receptor subunits that manifest their effects later in life, being shown as deficits in neuronal activity in the ...