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The primary causes include post-cataract surgery, certain medications, and, less commonly, neurological or ophthalmological conditions. Post-cataract surgery is a common cause, as replacing the natural lens with a synthetic one increases exposure to blue light, leading to temporary blue-tinted vision. This effect usually resolves as the eye adapts.
After cataract surgery, patients with diabetes mellitus are generally acknowledged to have an increased risk of macular edema. [ 12 ] A prior history of retinal vein occlusion was the only significant preoperative risk factor in a large retrospective series of 1659 consecutive cataract surgeries.
Cataract surgery is the most common application of lens removal surgery, and is usually associated with lens replacement. It is used to remove the natural lens of the eye when it has developed a cataract, a cloudy area in the lens that causes visual impairment. [4] [10] Cataracts usually develop slowly and can affect one or both eyes. [4]
Cataract surgery is the removal of the natural lens of the eye that has developed a cataract, an opaque or cloudy area. [3] Over time, metabolic changes of the crystalline lens fibres lead to the development of a cataract, causing impairment or loss of vision.
Disrupting cortical homeostatic processes after vision has been lost may prevent or setback the emergence of hallucinations. [10] At varying stages of the cortical grading, acetylcholine (ACh) may impact the balance of thalamic and intracortical inputs as well as the balance in between bottom-up and top-down. [10]
Posterior capsular opacification, also known as after-cataract, is a condition in which months or years after successful cataract surgery, vision deteriorates or problems with glare and light scattering recur, usually due to thickening of the back or posterior capsule surrounding the implanted lens, so-called 'posterior lens capsule opacification'.
PCO is a common side-effect of many cataract surgeries and is easily treatable with a one-time laser capsulotomy procedure (see below). Accommodating IOLs interact with ciliary muscles and zonules , using hinges at both ends to "latch on" and move forward and backward inside the eye using the same nerves and musculature as normal accommodation.
A minority of patients may regain low or moderate visual acuity, but over 70% are left clinically blind with worse than 20/400 vision. [4] A Nigerian study showed other complications include secondary glaucoma, hyphaema, and optic atrophy. [5] Couching does not compare favourably to modern cataract surgery.