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  2. Sleep Experts Share 7 Reasons You’re Sleeping So Much - AOL

    www.aol.com/sleep-experts-share-7-reasons...

    Many medication classes can cause excessive sleepiness or disrupt sleep, leading to EDS, say Dr. Winter and O’Malley. Those medications include: Antihistamines. Benzodiazepines.

  3. Excessive daytime sleepiness - Wikipedia

    en.wikipedia.org/wiki/Excessive_daytime_sleepiness

    EDS can affect the ability to function in family, social, occupational, or other settings. [1] [2] A proper diagnosis of the underlying cause and ultimately treatment of symptoms and/or the underlying cause can help mitigate such complications. [3] According to the National Sleep Foundation, around 20 percent of people experience EDS. [4]

  4. Hypersomnia - Wikipedia

    en.wikipedia.org/wiki/Hypersomnia

    Early hydrocephalus can also cause severe excessive daytime sleepiness. [16] Additionally, head trauma can be associated with a primary/central hypersomnia, and symptoms similar to those of idiopathic hypersomnia can be seen within 6–18 months following the trauma. However, the associated symptoms of headaches, memory loss, and lack of ...

  5. Lisinopril - Wikipedia

    en.wikipedia.org/wiki/Lisinopril

    Lisinopril is a medication belonging to the drug class of angiotensin-converting enzyme (ACE) inhibitors and is used to treat hypertension (high blood pressure), heart failure, and heart attacks. [7] For high blood pressure it is usually a first-line treatment.

  6. Which Antidepressants Can Cause ED? - AOL

    www.aol.com/antidepressants-cause-ed-105700786.html

    Abruptly stopping your medication can result in flu-like symptoms, such as nausea, headache, fatigue and other effects relating to antidepressant discontinuation syndrome.

  7. Idiopathic hypersomnia - Wikipedia

    en.wikipedia.org/wiki/Idiopathic_hypersomnia

    Idiopathic hypersomnia (IH) is a neurological disorder which is characterized primarily by excessive sleep and excessive daytime sleepiness (EDS). [1] Idiopathic hypersomnia was first described by Bedrich Roth in 1976, and it can be divided into two forms: polysymptomatic and monosymptomatic.

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