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In rats, after a 24-hour sleep deprivation, it was found that there was an increase of slow-wave activity in NREM sleep, [42] which corresponds directly with the human brain which when sleep deprived, prioritizes NREM sleep over REM sleep, implying that the NREM sleep is responsible for regulating and compensating for missed sleep. [43]
REM sleep is considered closer to wakefulness and is characterized by rapid eye movement and muscle atonia. NREM is considered to be deep sleep (the deepest part of NREM is called slow wave sleep), and is characterized by lack of prominent eye movement, or muscle paralysis. Especially during non-REM sleep, the brain uses significantly less ...
Sleep onset is the transition from wakefulness into sleep. Sleep onset usually transits into non-rapid eye movement sleep (NREM sleep) but under certain circumstances (e.g. narcolepsy ) it is possible to transit from wakefulness directly into rapid eye movement sleep (REM sleep).
This is particularly apparent in the right hemisphere. In non-sleep-deprived people involved in verbal learning and arithmetic tasks, the anterior cingulate cortex and the right prefrontal cortex are active. Following sleep deprivation, there is increased activation of the left inferior frontal gyrus and the bilateral parietal lobes. This ...
Sleep is divided into two broad types: non-rapid eye movement (non-REM or NREM) sleep and rapid eye movement (REM) sleep. Non-REM and REM sleep are so different that physiologists identify them as distinct behavioral states. Non-REM sleep occurs first and after a transitional period is called slow-wave sleep or deep sleep.
Sample hypnogram showing one sleep cycle (the first of the night) from NREM through REM. The sleep cycle is an oscillation between the slow-wave and REM (paradoxical) phases of sleep. It is sometimes called the ultradian sleep cycle, sleep–dream cycle, or REM-NREM cycle, to distinguish it from the circadian alternation between sleep and ...
It occurs during stage 2 NREM sleep. It is the "largest event in healthy human EEG". [1] They are more frequent in the first sleep cycles. K-complexes have two proposed functions: [1] first, suppressing cortical arousal in response to stimuli that the sleeping brain evaluates not to signal danger, and second, aiding sleep-based memory ...
REM sleep is decreased during the first half of the sleep period and stage 1 sleep is increased in the second half of the sleep period. [5] Most antidepressants, in particular selective serotonin re-uptake inhibitors (SSRIs), such as citalopram and paroxetine, are potent inhibitors of REM sleep and may also cause a REM rebound on discontinuation.